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Call for PapersGeroScience is issuing a call for submissions for special themed issues: "Aging in Companion Animals” Associate Editors Dr. Silvan Urfer, Dr. Jessica Hoffman and Dr. Matt Kaeberlein and the editorial team of GeroScience (the Journal of the American Aging Association) invite submissions of original research articles, opinion papers and review articles related to research focused on using companion animals as models in all aspects of aging research. While we have made tremendous progress in aging research using laboratory animals, these studies have been done in tightly controlled environments, with minimal environmental and genetic variation. Privately owned companion animals are an exciting emerging model in that they share the highly variable human environment and its risk factors, spontaneously develop many of the same age-related diseases that are associated with normative aging in humans, receive comparable levels of medical care, and generally have excellent veterinary medical data available. In addition, companion dogs and cats are divided into a genetically diverse mixed breed population, as well as a population of purebred animals that represent hundreds of genetically distinct, inbred strains that present an ideal opportunity to model genotype-environment interactions. Companion dogs in particular are also highly phenotypically diverse, opening additional opportunities to study the effects of various phenotypes and their underlying genotypes on normative aging and age-related disease. This call-for-papers is aimed at providing a platform for the more widespread use of companion animals as models in geroscience, with the goal of being able to better model both the genetic diversity as well as the environmental variation found in the human population. This includes lifespan and causes of death in various genetic and environmental backgrounds, as well as studies focusing on individual diseases, comparative epidemiology, and One Health aspects within a diverse genetic and environmental background. We are also interested in manuscripts investigating interventional approaches to aging and age-related diseases, leveraging the fact that companion animals age at roughly 7-10 times the human rate, and encourage submissions to develop them as models and potential sentinels for human health. All manuscripts accepted from this Call for Papers will be included in a unique online article collection to further highlight the importance of this topic. All manuscripts should be submitted online here: https://www.editorialmanager.com/jaaa/default.aspx Please indicate during the submission process by checking the relevant box that the manuscript is submitted in response to this Call for Papers. Manuscripts will undergo normal peer review as they are received. Manuscripts will be published online as they are accepted. Articles published from this Call for Papers will be highlighted with a special “Call for Papers” banner on the article PDF, as well as included in the "Aging in Companion Animals” online article collection at time of publication. Submission of cover image ideas is encouraged. The submission of manuscripts in response to this Call for Papers is continuous. If you have any question please contact Dr. Silvan Urfer via email at [email protected].
“White matter lesions in age-related cognitive decline: etiology, risk factors, prevention and repair“ Associate Editors Drs. Anna Csiszar and Farzaneh Sorond along with the Editor-in-Chief Dr. Zoltan Ungvari, and the editorial team of GeroScience (Journal of the American Aging Association) in collaboration with the newly established “Albert Research Institute for White Matter and Cognition” (https://albertwhitemattterresearchinstitute.org) invite submission of original research articles, opinion papers and review articles related to research focused on understanding the role of white matter disease in age-related cognitive decline. White matter hyperintensities (WMH) on brain MRI are perpetual findings in an aging brain, and strongly associated with cardiovascular risk factors, cognitive decline and dementia. Yet, our mechanistic understanding of the white matter lesion etiology in dementia is entirely unexploited and poorly understood. This call-for-papers is aimed at providing a platform for the dissemination of critical novel ideas related to brain white matter injury and repair mechanisms, which are critical to the functional interplay between neurons, vessels, and glial cells (neurovascular unit) and the plasticity of the myelinated axons, and ultimately brain health across life span. We welcome manuscripts focusing on preclinical models of adaptive myelination, neurodegeneration, neurovascular inflammation and cerebrovascular regulation, targeting the role of endothelium, pericytes, microglia, oligodendrocytes, the neurovascular unit and the oligovascular niche. Similarly, human clinical, biomarker and imaging studies exploring these mechanisms in age-related cognitive declines are also invited. Manuscripts exploring the causal role of molecular mechanisms of white matter plasticity and repair as they are related to lifestyle (e.g., diet, exercise, smoking), medical (e.g. blood pressure, cancer treatments), or environmental exposures are of particular interest. Authors are also encouraged to submit manuscripts focusing on translational aspects of brain white matter and cognition research. All manuscripts accepted from this Call for Papers will be included in a unique online article collection to further highlight the importance of this topic. All manuscripts should be submitted online here: https://www.editorialmanager.com/jaaa/default.aspx Please indicate during the submission process by checking the relevant box that the manuscript is submitted in response to this Call for Papers. Manuscripts will undergo normal peer review as they are received. Manuscripts will be published online as they are accepted. Articles published from this Call for Papers will be highlighted with a special “Call for Papers” banner on the article PDF, as well as included in the " White matter lesions in age-related cognitive decline” online article collection at time of publication. Submission of cover image ideas is encouraged. The submission of manuscripts in response to this Call for Papers is continuous. If you have any question please contact Dr. Farzaneh Sorond ([email protected]) or Dr. Anna Csiszar ([email protected]) via email. Previous Calls:"Understanding the impact of aging on the susceptibiity and response to COVID-19 infection" "Understanding and overcoming the mechanisms driving age-related sarcopenia" Please see the detailed announcements for each here: “Understanding Senescence in Brain Aging and Alzheimer's Disease “ Guest Editors Drs. Julie Andersen and Darren Baker, Associate Editor Dr. Anna Csiszar and Editor-in-Chief Dr. Zoltan Ungvari, and the editorial team of GeroScience (Journal of the American Aging Association; 2018 Impact Factor: 6.44) invite submission of original research articles, opinion papers and review articles related to research focused on understanding the role of senescence in brain aging and in Alzheimer's disease. Senescent cells accumulate in aging and pathological conditions associated with accelerated aging. While earlier investigations focused on cellular senescence in tissues and cells outside of the brain (e.g. adipose tissue, dermal fibroblasts, cells of the cardiovascular system), more recent studies started to explore the role of senescent cells in age-related decline of brain function and the pathogenesis of neurodegenerative disease and vascular cognitive impairment. This call-for-papers is aimed at providing a platform for the dissemination of critical novel ideas related to the functional and physiological consequences of senescence in diverse brain cell types (e.g., oligodendrocytes, pericytes, astrocytes, endothelial cells, microglia, neural stem cells), with the ultimate goal to identify novel targets for treatment and prevention Alzheimer's disease, Parkinson's disease and vascular cognitive impairment. We welcome manuscripts focusing on senescent-cell-targeting mouse models, the role of paracrine senescence, senescence pathways in terminally differentiated neurons, the pleiotropic effects of systemic senescence, the role of senescence in neuroinflammation and the protective effects of senolytic therapies. We are especially interested in manuscripts exploring the causal role of molecular mechanisms of aging in induction of cellular senescence as well as links between lifestyle (e.g., diet, exercise, smoking), medical treatments (e.g. cancer treatments), exposure environmental toxicants and cellular senescence in the brain. We encourage submission of manuscripts on developing innovative strategies to identify and target senescent cells for prevention/treatment of age-related diseases of the brain. Authors are also encouraged to submit manuscripts focusing on translational aspects of senescence research. All manuscripts accepted from this Call for Papers will be included in a unique online article collection to further highlight the importance of this topic. All manuscripts should be submitted online here: https://www.editorialmanager.com/jaaa/default.aspx Please indicate during the submission process by checking the relevant box that the manuscript is submitted in response to the Call for Papers “Understanding Senescence in Brain Aging and Alzheimer's Disease”. Manuscripts will undergo normal peer review as they are received. Manuscripts will be published online as they are accepted. Articles published from this Call for Papers will be highlighted with a special “Call for Papers” banner on the article PDF, as well as included in the "Understanding Senescence in Brain Aging and Alzheimer's Disease” online article collection at time of publication. Submission of cover image ideas is encouraged. The submission of manuscripts in response to this Call for Papers is continuous. If you have any question please contact Dr. Zoltan Ungvari via email at [email protected] . Editor-in-Chief: Zoltan Ungvari, MD, PhD ISSN: 0161-9152 (print version); ISSN: 1574-4647 (electronic version) Springer Netherlands For submission information click here. To subscribe to our "Table of Contents" updates click here. Inquiries please email [email protected] GeroScience is a quarterly, international, peer-reviewed journal that publishes articles related to research in the biology of aging and research on biomedical applications that impact aging. The scope of articles to be considered include evolutionary biology, biophysics, genetics, genomics, proteomics, molecular biology, cell biology, biochemistry, endocrinology, immunology, physiology, pharmacology, neuroscience, and psychology. Articles concerning clinical studies will also be considered if the results relate to underlying biological mechanisms of aging. Such studies should reflect more than issues related to the care and treatment of geriatric patients. Papers concerned with social, economic, and political issues of aging will generally not be considered unless they relate directly to biomedical gerontology. In addition to manuscripts emerging from original research, the journal actively solicits research reviews of important topics in biomedical gerontology. Other types of manuscripts are also acceptable, such as commentaries, debates, and meeting reports. GeroScience is a bi-monthly, international, peer-reviewed journal that publishes articles related to research in the biology of aging and research on biomedical applications that impact aging. The Journal is available online to current Association scientific members. Lay members and student members can purchase online subscriptions for $30 per year. For more information on GeroScience, please review the information at the publisher's site by clicking here. EDITORIAL BOARD
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